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Clinical and pathologic studis show that lvatd plasma lvls total cholstol (total-C), LDL-cholstol (LDL-C) and apolipopotin B (apB) pomot human athosclosis and a isk actos dvloping cadiovascula disas, whil incasd lvls HDL-cholstol (HDL-C) a associatd with a dcasd cadiovascula isk. Lik LDL, cholstol-nichd TG-ich lipopotins, including VLDL, IDL and mnants, can alspomot athosclosis. lvatd plasma TGs a quntly ound in a tiad with low HDL-C lvls and small LDL paticls, as wll as in association with non-lipid mtabolic isk actos coonay hat disas (CHD). As such, total plasma TG has not consistntly bn shown tb an indpndnt isk actCHD. uthmo, th indpndnt ct aising HDL lowing TG on th isk coonay and cadiovascula mobidity and motality has not bn dtmind.

Colchicin: Cass myopathy, including habdomyolysis, hav bn potd with atovastatin co-administd with colchicin, and caution should b xcisd whn pscibing ATOLIP CV with colchicin.

Gapuit Juic: Contains on mcomponnts that inhibit CYP3A4 and can incas plasma concntations atovastatin, spcially with xcssiv gapuit juic consumption (>1.2 lits/day).

Th dosag clopidogl in unstabl angina (UA) / Non-ST-lvatd myocadial inaction (NSTMI) is 75 mg daily at a singl loading dos. In patints with STMI, cnt MI, stok piphal aty disas, th commndd dos clopidogl is 75 mg onc daily.

A cass habdomyolysis with acut nal ailu sconday tmyoglobinuia hav bn potd with atovastatin and with oth dugs in this class. A histoy nal impaimnt may b a isk actth dvlopmnt habdomyolysis. Such patints mit clos monitoing skltal muscl cts.

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atorlip and atorvastatin-metabolites are substrates of the oatp1b1 transporter.